Omicron and Long Covid
Long Covid and Omicron SARS-CoV-2 can spread from cell to cell through cell walls. This is a way of hiding from the immune system. This ability was limited in SARS-CoV-1. SARS-CoV-1 leaves the infected cell and infects new cells through receptors. This is the classic way. This leaves the virus more vulnerable to immune system attacks and antibody neutralization. This feature of SARS-CoV-2 is very important in terms of hiding it from the immune response.. Variants from SARS-CoV-2 and SARS-CoV-2 use ACE2 and TMPRSS2 receptors to infect cells. However, with the Omicron BA.1, this situation has changed. TMPRSS2 is an abundant receptor in the lung. Omicron has a lower affinity for the TMPRSS2 receptor than other variants.( 5 times less than Delta). This explains why Omicron BA.1 has less lung pathogenicity than Delta. In addition, Omicron can enter the cell directly by the endosomal route without ACE2 and TMPRSS2. This ability was almost non-existent in previous variants. And Omicro